Non Convulsive Status Epileptics in a Child with Acute Organophosphorus Poisoning: A Case Report

Organophosphate (OP) poisoning is one of the most common toxicological emergencies, especially in developing countries. They work by inhibiting cholinesterase enzyme lead to severe cholinergic toxicities. Organophosphorus intoxication occurs after cutaneous exposure, respiratory inhalation or gastrointestinal tract absorption. Exposure to organophosphate and other agents increases yearly, with up to 300,000 fatalities [1,2]. Acute toxicity from organophosphorous presents with manifestations of cholinergic excess. Primary toxic effects involve the autonomic nervous system, neuromuscular junction, and central nervous system (CNS) [3].